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Muscular Dystrophy
X-Linked
  • Description
  • Signalment
  • Clinical Features
  • Neurolocalization
  • Genetics
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Muscular dystrophies are examples of degenerative myopathic conditions of both dogs and cats. These diseases are exemplified by an X-linked disease of golden retrievers and other breeds. In humans, this type of muscular dystrophy is referred to as Duchenne's muscular dystrophy.  These disease processes result from gene-associated abnormalities of a muscle membrane protein (dystrophin).
Age of Onset: 6-9 weeks of age
​

Sex Predisposition: Males are more commonly affected
Clinical Course:
​Clinical signs may plateau after 6 months of progressive deterioration.
​

Clinical Signs:
Behavioral/Mental Awareness
Exercise intolerance

Posture and Appearance
Plantigrade stance
Kyphosis to lordosis
​Failure to grow


Movement
Weakness
“Stiffness”
“Shuffling” gait
Abduction of elbows
Bunny-hopping gait
Exercise-intolerance


Proprioception
Normal proprioception but poor hopping response

Cranial Nerves
Enlargement of the base of the tongue
Inability to fully open the jaw (trismus)
Weak barking/dysphonia

Special Functions (e.g. respiration; urination)
Hypersalivation
Pharyngeal and esophageal dysfunction

Muscle Atrophy
Generalized muscle atrophy (head and limb muscles)
Generalized muscle hypertrophy

Other

Mineralization of the tongue
​
​Diffuse neuromuscular disease
X-linked recessive mutation in the dystrophin gene

For breed specific genetic testing, follow the links below:
​https://www.genimal.com/dna-tests/dog/muscular-dystrophy-in-brittany-spaniel-variant-2-dmd/
To read more about this disease click below:
References
Alpha2-laminin Deficiency
  • Description
  • Signalment
  • Clinical Features
  • Neurolocalization
  • Genetics
<
>
Muscular dystrophies are examples of degenerative myopathic conditions of both dogs and cats.  These disease processes result from gene-associated abnormalities of a muscle membrane protein (dystrophin, sarcoglycan, or laminin).
Age of Onset: First 4 months of life
​

Sex Predisposition:  Any sex of animal can be affected 
Clinical Course:
​Clinical signs may plateau after 6 months of progressive deterioration.
​

Clinical Signs:
Behavioral/Mental Awareness
Exercise intolerance

Posture and Appearance
Plantigrade stance
Kyphosis to lordosis
​Failure to grow


Movement
Weakness
“Stiffness”
“Shuffling” gait
Abduction of elbows
Bunny-hopping gait
Exercise-intolerance


Proprioception
Normal proprioception but poor hopping response

Cranial Nerves
Enlargement of the base of the tongue
Inability to fully open the jaw (trismus)
Weak barking/dysphonia

Special Functions (e.g. respiration; urination)
Hypersalivation
Pharyngeal and esophageal dysfunction

Muscle Atrophy
Generalized muscle atrophy (head and limb muscles)
Generalized muscle hypertrophy

Other

Mineralization of the tongue
​
​Diffuse neuromuscular disease
Autosomal recessive mutation in the alpha-2 laminin gene
​To read more about this disease click below:
References
To read more about this disease click below:
References
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