Laryngeal Paralysis (Congenital)
Congenital laryngeal paralysis occurs through neurodegeneration of the recurrent laryngeal nerve which innervates the cricoarytenoideus dorsalis muscle resulting in the failure of the arytenoid cartilages to abducting normally during inspiration.
Age of Onset: Less than 3 years of age
Sex Predisposition: Any sex of animal can be affected; Males were reported to be more commonly affected than females (Gaber et. al., 1985)
Clinical Course:
Clinical signs may worsen over time
Clinical signs may worsen over time
Clinical Signs:
Movement
Collapse
Weakness
Special Functions (e.g. respiration; urination)
Dyspnea
Resting tachypnea
Exercise intolerance
Inspiratory stridor
Muscle Atrophy
Atrophy of cricoarytenoideus dorsalis - not apparent on clinical examination
Other
Cough
Hoarse bark (dysphonia)
Noisy breathing (wheezing)
Vomiting/regurgitation
Movement
Collapse
Weakness
Special Functions (e.g. respiration; urination)
Dyspnea
Resting tachypnea
Exercise intolerance
Inspiratory stridor
Muscle Atrophy
Atrophy of cricoarytenoideus dorsalis - not apparent on clinical examination
Other
Cough
Hoarse bark (dysphonia)
Noisy breathing (wheezing)
Vomiting/regurgitation
Neuromuscular junction
Autosomal dominant inheritance
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